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INSERM U397, Institut L. BUGNARD (R.E., S.C., J.-F.A., F.B.), 31054 Toulouse Cédex, France; INSERM U325 et SERLIA, Institut Pasteur (C.F.), 59019 Lille Cédex, France; Department of Pathology (C.R.-A., G.S.G.), University of Chicago, Chicago, Illinois 60637
Address all correspondence and requests for reprints to: Francis Bayard, Institute Louis Bugnard, Chu Rangueil, INSERM U397, 1 avenue Jean Poulhes, Toulouse, France 31054.
Estradiol significantly decreases fatty streak formation in the aortic root of chow-fed apolipoprotein E-deficient mice. In contrast, immunodeficient mice with homozygous disruption at the recombinase activating gene 2 loci present fatty streak development that is insensitive to estradiol. Lymphocytes thus appear to be required for development of the atheroprotective effect of estradiol in this mouse model.
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